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PANT, VINOD
One or more keywords matched the following items that are connected to PANT, VINOD
Item TypeName
Academic Article p53-mediated senescence impairs the apoptotic response to chemotherapy and clinical outcome in breast cancer.
Concept Tumor Suppressor Protein p53
Academic Article Heterodimerization of Mdm2 and Mdm4 is critical for regulating p53 activity during embryogenesis but dispensable for p53 and Mdm2 stability.
Academic Article The p53-Mdm2 feedback loop protects against DNA damage by inhibiting p53 activity but is dispensable for p53 stability, development, and longevity.
Academic Article Spontaneous tumorigenesis in mice overexpressing the p53-negative regulator Mdm4.
Academic Article A high-frequency regulatory polymorphism in the p53 pathway accelerates tumor development.
Academic Article The p53 pathway in hematopoiesis: lessons from mouse models, implications for humans.
Academic Article Limiting the power of p53 through the ubiquitin proteasome pathway.
Academic Article Differential regulation of E2F transcription factors by p53 tumor suppressor protein.
Academic Article The ups and downs of p53 regulation in hematopoietic stem cells.
Academic Article Dissecting the p53-Mdm2 feedback loop in vivo: uncoupling the role in p53 stability and activity.
Academic Article The p53 inhibitor Mdm4 cooperates with multiple genetic lesions in tumourigenesis.
Academic Article Synergistic and additive effect of retinoic acid in circumventing resistance to p53 restoration.
Academic Article Daxx Functions Are p53-Independent In Vivo.
Academic Article Somatic Trp53 mutations differentially drive breast cancer and evolution of metastases.
Academic Article Transient enhancement of p53 activity protects from radiation-induced gastrointestinal toxicity.
Academic Article Alterations of the Mdm2 C-Terminus Differentially Impact Its Function In Vivo.
Academic Article Differential Gain-of-Function Activity of Three p53 Hotspot Mutants In Vivo.
Academic Article Dimeric p53 Mutant Elicits Unique Tumor-Suppressive Activities through an Altered Metabolic Program.
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  • Tumor Suppressor Protein p53